© Dr. Sabine Hombach Research 2014
Dr. S. Hombach, M.D., Ph.D.
Biography
Dr. S. Hombach, M.D., Ph.D.
Associate Professor
Section Head, Gross Anatomy
Department of Human Anatomy
and Cell Science
Professional Affiliation
Biography
Dr. Hombach-Klonisch completed her medical training at the Justus-
Liebig University in Giessen, Germany, in 1991. She pursued her
PhD training in Pathophysiology at the University in Giessen under
the supervision of Professor Dr. Werner Seeger and received her
PhD in 1994.
After spending family time with her small children, Dr. Hombach was
granted a post-doctoral fellowship (1998-2000) by the Province of
Saxony-Anhalt, Germany, and performed research on the role of
relaxin-like peptides in the placenta at the Department of Anatomy
and Cell Biology, Martin-Luther University Halle-Wittenberg. In
2000, Dr. Hombach accepted a position as Assistant Professor at
the University Halle-Wittenberg and focused her research on cell
migration in human breast and thyroid cancer. For her pioneer work
on identifying relaxin-like peptides as novel paracrine factors in
thyroid cancer cell migration and invasiveness Dr. Hombach was
awarded the Merck European Thyroid von Basedow Research Prize
from the German Endocrine Society in 2002. In 2003, Dr. Hombach
received her professional degrees as Anatomist by the German
Anatomical Society and the Medical Association of Saxony-Anhalt.
She completed her Habilitation in Tumor Biology in 2004 at the
Martin-Luther-University Halle-Wittenberg under the guidance of
Professor Dr. Dr. B. Fischer. In 2004, she was recruited to the
Department of Anatomy and Cell Science, Faculty of Medicine, at
the University of Manitoba. Her professional engagement continues
to be in anatomy teaching and cancer research for which she
received the Murray L. Barr Young Investigator Award by the
Canadian Association for Anatomy, Neurobiology and Cell Biology in
2009. As an Associate Professor, Dr. Hombach’s current research
investigates molecular mechanisms utilized by heterogeneous
cancer cell populations to evade or overcome chemotherapeutic
insult and promote cancer cell survival and tissue invasion.