St. John's Wort? Though it may help with depression, couldn't find anything for panic attacks.

Breathing retraining in the treatment of panic disorder: Efficacy, caveats and indications. .
    AUTHOR: Taylor,-Steven
    SOURCE: Scandinavian-Journal-of-Behaviour-Therapy. 2001; Vol 30(2): 49-56.
    Full Text: EBSCO Online
    PUBLISHER: Norway: Scandinavian Univ Press.
    ISSN: 0284-5717
    PUBLICATION YEAR: 2001
    ABSTRACT: Breathing retraining is commonly used in the treatment of panic disorder. Although several
    studies suggest that this intervention is effective in reducing panic frequency, concerns have been raised
    about its routine use. For example, recent research suggests that hyperventilation plays a limited role in
    producing panic attacks. This suggests that breathing retraining may only be useful for a minority of
    patients, for whom hyperventilation (or chest breathing) plays a role in producing panic symptoms. A
    further concern is that breathing retraining may be counterproductive to the extent that it prevents patients
    from learning that their catastrophic beliefs are unfounded. This article reviews the evidence for the efficacy
    of breathing retraining and considers the caveats and indications for its use. It is concluded that breathing
    retraining can play a useful role in the treatment of panic disorder, although clinicians must exercise care to
    ensure that it is not misused by patients as a means of escaping or avoiding feared sensations.

The treatment of panic disorder: A comparative study between breathing retraining and cognitive behavioral therapy.
    AUTHOR: Berger,-Beth-Creager
    SOURCE: Dissertation-Abstracts-International:-Section-B:-The-Sciences-and-Engineering. 2001 Mar;
    Vol 61(8-B): 4390.
    ISSN: 0419-4217
    PUBLICATION YEAR: 2001
    UMI ORDER NUMBER: AAI9984306
    ABSTRACT: Cognitive Behavioral Therapy (CBT) is a widely used and highly effective treatment for
    panic disorder. It consists of different components and breathing retraining (BR) is often included. It is not
    clear what mechanism of panic is affected when panic symptoms decrease. There has been an increase in
    studies over the past ten years looking at the respiratory component of panic. It is thought that BR may be
    effective in the treatment of panic if the respiratory aspect is a primary element in panic. Few studies have
    looked solely at breathing retraining in the treatment of panic. A treatment outcome study was conducted
    to assess if BR may be as effective as CBT in decreasing the frequency and severity of panic symptoms.
    Ten people with panic disorder were randomly assigned to CBT without BR and 11 were randomly
    assigned to a BR group. All participants kept a panic attack journal 4 weeks prior to starting treatment
    through 4 weeks after treatment. The BR group included six 30 to 60 minute sessions of breathing
    retraining. The CBT group followed a modified version of Craske, Barlow, and Meadows protocol
    (1994) which consisted of ten 45 to 90 minute sessions. Results show that BR appears as effective as
    CBT in the treatment of panic disorder. There were no significant pre to post differences in the severity or
    frequency of panic attacks between the two groups. It is felt that the respiratory component is more
    essential in panic than has been acknowledged in the past. Because breathing retraining can be taught more
    rapidly than cognitive behavioral therapy and therefore provide relief sooner, it can be a useful tool in the
    treatment of panic.

Efficacy of usual antidepressant dosing regimens of fluoxetine in panic disorder: Randomised, placebo-controlled trial.
    AUTHOR: Michelson,-David; Allgulander,-Christer; Dantendorfer,-Karl; Knezevic,-Aleksandar;
    Maierhofer,-Dagmar; Micev,-Vitomar; Paunovic,-Vladimir-R; Timotijevic,-Ivana; Sarkar,-Neena;
    Skoglund,-Lennart; Pemberton,-S-Craig
    SOURCE: British-Journal-of-Psychiatry. 2001 Dec; Vol 179: 514-518.
    Full Text: HighWire
    PUBLISHER: England: Royal Coll of Psychiatrists.
    ISSN: 0007-1250
    PUBLICATION YEAR: 2001
    ABSTRACT: Assessed the efficacy of the usual antidepressant dose of fluoxetine in treating full panic
    attacks. 180 patients with panic disorder were randomised to placebo or to fluoxetine initiated at 10 mg
    daily for 1 week and then increased to 20 mg daily. The trial lasted 12 weeks, but after 6 weeks patients
    who had failed to achieve a satisfactory response were eligible for dose escalation to a maximum of 60 mg
    of fluoxetine daily. Fluoxetine was associated with a statistically significantly greater proportion of
    panic-free patients compared with placebo after 6 weeks and at end-point. Fluoxetine at a dose of 20 mg
    daily is safe and efficacious in reducing symptoms of panic disorder. Patients who fail to obtain a
    satisfactory response at 20 mg daily may benefit from further dose increases.

Prevention of panic.
    AUTHOR: Gardenswartz,-Cara-Ann
    SOURCE: Dissertation-Abstracts-International:-Section-B:-The-Sciences-and-Engineering. 2001 Aug;
    Vol 62(2-B): 1077.
    ISSN: 0419-4217
    PUBLICATION YEAR: 2001
    UMI ORDER NUMBER: AAI3005975
    ABSTRACT: Over the past 30 years, effective psychological treatments for anxiety disorders have been
    developed and empirically tested. In particular, cognitive-behavioral treatment of panic disorder has been
    shown to be both statistically and clinically significant. However, little research has looked at primary or
    secondary prevention of panic disorder. The goal of this study was to use our knowledge about effective
    treatment strategies for panic disorder to implement and empirically test a prevention program designed to
    diminish panic symptomatology and prevent panic disorder. Participants who had a recent panic attack
    were randomly assigned to either a one-day prevention workshop group or a waitlist control group.
    Participants were followed for six months post-workshop in order to determine whether the workshop
    was effective in decreasing the anxiety and panic symptomatology and/or panic disorder during this period.
    Results indicated that the prevention workshop was successful in reducing both panic disorder diagnoses,
    and frequency and intensity of panic attacks. Furthermore, the workshop decreased social avoidance,
    and avoidance in the participants.

Prospective evaluation of the efficacy of a brief cognitive-behavioral intervention on the
development of panic disorder and anxiety in a high-risk, nonclinical college population.
    AUTHOR: Abplanalp,-Bart-Solomon
    SOURCE: Dissertation-Abstracts-International:-Section-B:-The-Sciences-and-Engineering. 2001 Sep;
    Vol 62(3-B): 1562.
    ISSN: 0419-4217
    PUBLICATION YEAR: 2001
    UMI ORDER NUMBER: AAI3008262
    ABSTRACT: The present study sought to determine the effectiveness of a brief cognitive-behavioral
    intervention (called Panic Prevention Training) in lowering anxiety sensitivity (a proposed risk factor in the
    development of panic) and subsequently reducing the risk of developing panic attacks and anxiety-related
    symptomatology. The short-term effects of the intervention in lowering anxiety sensitivity (AS) were
    examined through a battery of self-report measures. In addition, participants inhaled a 35% CO2/65% O
    2 gas mixture designed to generate intense physiological sensations. The long-term effects of the
    intervention on the incidence of panic attacks and panic-related apprehension were then assessed with a
    follow-up interview one year after completion of the initial phase of the study. Study participants were 405
    university students with elevated anxiety sensitivity (high AS). Participants were assigned to either (1) the
    Panic Prevention Training (PPT) group or (2) a Non-Specific Training (NST) group. The purpose of the
    PPT group was to provide detailed information regarding panic anxiety and to habituate participants' fears
    through cognitive restructuring and repeated exposure exercises. The NST condition was designed to
    control for more general factors associated with participation in a group setting. The intervention
    demonstrated significant short-term effects with PPT participants showing a reduction in both the specific
    factors related to anxiety sensitivity and one general measure of psychopathology and mood. In addition,
    the intervention was successful at reducing fear and self-reported panic in response to the CO2. At
    follow-up, the groups did not differ in either panic attack or panic disorder incidence. However, among
    those participants who did experience a panic attack during the follow-up period, the PPT group showed
    significantly lower panic-related apprehension compared to the NST group. The current study provides
    support for the short-term effects of a cognitive-behavioral intervention at lowering anxiety sensitivity and
    extends the work of previous studies in showing that these effects are maintained at one-year follow-up.
    Although the intervention failed to affect the incidence rate for panic attacks or panic disorder, it was
    successful at significantly lowering panic-related apprehension. The study provides preliminary support for
    the effectiveness of Panic Prevention Training to reduce both proximal and distal risk factors in the
    development of panic disorder.

Reboxetine, a selective norepinephrine reuptake inhibitor, is an effective and well-tolerated treatment for panic disorder.
    AUTHOR: Versiani,-Marcio; Cassano,-Giovanni; Perugi,-Giulio; Benedetti,-Alessandra; Mastalli,-Luigia;
    Nardi,-Antonio; Savino,-Mario
    SOURCE: Journal-of-Clinical-Psychiatry. 2002 Jan; Vol 63(1): 31-37.
    PUBLISHER: US: Physicians Postgraduate Press.
    ISSN: 0160-6689
    PUBLICATION YEAR: 2002
    ABSTRACT: Tricyclic antidepressants and selective serotonin reuptake inhibitors (SSRIs) as well as
    benzodiazepines have been shown to be effective for the treatment of panic disorder. The aim of this study
    was to determine the efficacy and tolerability of reboxetine, a novel selective norepinephrine reuptake
    inhibitor, in patients with panic disorder with and without agoraphobia. 82 patients (aged 18-65 years)
    with Mental Disorders-III-Revised (DSM-III-R) panic disorder, with or without agoraphobia, were
    randomly assigned to receive 6 to 8 mg/day of reboxetine (42 patients) or placebo (40 patients) for 8
    weeks in this placebo-controlled, parallel-group, double-blind clinical trial. Of the 82 patients enrolled in
    the trial, 75 were considered in the analysis (37 patients in the reboxetine group and 38 patients in the
    placebo group). At last assessment, there was a significant reduction in the mean number of panic attacks
    (range, 9.3-1.2) and phobic symptoms (range, 8.1-3.2) in the reboxetine group compared with the
    placebo group (ranges, 8.5-5.8 and 7.7-5.2, respectively). Conclusions suggest reboxetine was effective
    and well tolerated in the treatment of panic disorder.

Early-onset fearful panic attack: A possible prodrome of early-onset severe psychopathology.
    AUTHOR: Goodwin,-Renee-D; Hamilton,-Steven-P
    SOURCE: Comprehensive-Psychiatry. 2002 Jan-Feb; Vol 43(1): 22-27.
    PUBLISHER: US: WB Saunders.
    ISSN: 0010-440X
    PUBLICATION YEAR: 2002
    ABSTRACT: To determine whether early-onset panic attack with fear at onset represents a possible
    prodrome of early-onset severe psychopathology, we drew data from the National Comorbidity Survey
    (N=8,098), a probability sample drawn from respondents aged 18-54 yrs in the US. Multivariate logistic
    regression analyses were used to determine differences in age of onset of comorbid mental disorders,
    familial psychopathology, and lethality of suicide attempts between those with early-onset panic attacks
    with fear at onset compared with individuals with panic attacks that do not meet this criteria. Logistic
    regression analyses revealed that respondents with early-onset panic attack with fear at onset had
    significantly earlier onset of almost every comorbid mental disorder, higher rates of familial
    psychopathology, and increased risk and lethality of suicide attempt compared with those with other panic
    attacks. These findings provide preliminary evidence to suggest that early-onset fearful panic attack may
    be a marker of risk of a lengthy course of severe psychopathology, which is predicted by familial mental
    illness, and is characterized by early onset and increased risk of serious suicidal behavior.

Hyperventilation challenge test in panic disorder and depression with panic attacks.
    AUTHOR: Nardi,-Antonio-E; Valenca,-Alexandre-M; Nascimento,-Isabella; Zin,-Walter-A
    SOURCE: Psychiatry-Research. 2001 Dec; Vol 105(1-2): 57-65.
    Full Text: ScienceDirect (tm)
    PUBLISHER: US: Elsevier Scientific.
    ISSN: 0165-1781
    PUBLICATION YEAR: 2001
    ABSTRACT: Determined whether panic disorder (PD) patients, major depressive patients without panic
    attacks (MD) and major depressive patients with panic attacks (MDP) respond similarly to
    hyperventilation challenge tests. Ss were 35 PD patients, 33 MDP patients, 27 MD patients and 30
    normal volunteers with no family history of anxiety or mood disorder. The patients had not been treated
    with psychotropic drugs for at least 1 week. They were induced to hyperventilate (30 breaths/min) for 4
    min, and anxiety was assessed before and after the test. A total of 16 (45.7%) PD patients, 12 (36.4%)
    MDP patients, 4 (11.1%) MD patients, and 2 (6.7%) normal volunteers had a panic attack after
    hyperventilating. The PD and MDP patients were significantly more responsive to hyperventilation than the
    MD patients and the normal volunteers. The MD patients had a significantly lower heart-rate response to
    the test than all the other groups. There is growing evidence that PD patients are more sensitive to the
    vasoconstrictive effects on basilar arterial blood flow caused by hyperventilation-induced hypocapnia than
    are comparison subjects. Data suggest that there is an association between panic attacks and
    hyperreactivity to an acute hyperventilation challenge test.